Chow Chow Dog Breed: A Breed that is Well-mannered and dignified, with a heart of gold. Breed Information, Characteristics, Common Health Problems, Recommended Exercise, Nutrition & Lifestyle.
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After more than two days of meeting, provincial health ministers and the federal health minister have been unable to come up with next steps on health care funding.

In a statement from the Council of the Federation, released while the ministers were still meeting, the organization representing premiers expressed disappointment with the lack of a federal response on the critical issue of sustainable health funding.

The provinces have been asking the federal government to agree to a meeting that would include Prime Minister Justin Trudeau and the premiers.

Following Tuesday’s discussions, a news conference led by B.C. Health Minister Adrian Dix was held.

For more info, please go to: https://globalnews.ca/news/9260436/healthcare-federal-funding-provinces/

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January is National Blood Donor Awareness Month, and 13-year-old Tymia Green is encouraging more people to donate. Tymia has sickle cell anemia and experiences pain crises that feels like “a knife is stabbing in and out” she said. Doctors compare the pain to a heart attack. A blood transfusion is the only thing that stops the pain and helps her get back to being a kid. When people donate blood, Tymia says “they’re giving me a life.” InsideEdition.com’s Johanna Li has more. #InsideEdition
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Check out ESNTLS, they sell the best essentials in the world: https://www.esntls.co/
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OH HEY! Welcome back. 🙂 Today I’m sharing my healthy summer morning routine with you guys.

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This video is about my tips and first steps for how to start a successful blog in 2021.

For the 2022 video: https://www.youtube.com/watch?v=m7Jw3a7CpNA

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Hope this was helpful! Happy blogging xo

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#blogging #bloggingtips #howtostartablog

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This video is giving you the top blogging tips for how to start a blog to make money.

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This video is all about the best blogging tips on how to start a blog and make money #howtostartablog #bloggingtips #howtostartablogtomakemoney #howtostartablogandmakemoney #blogging

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Inflammatory Bowel Disease Clinical essentials – Dr. Kiran Peddi MRCP(UK), FRCP(London), CCT(Gastro)

Inflammatory bowel disease (IBD) is an idiopathic chronic relapsing inflammation of the bowel that presents with recurrent episodes of abdominal pain and diarrhea. There are two sub-types of IBD: Crohn’s disease and ulcerative colitis.

Interestingly, while smoking is associated with an increased risk of Crohn’s disease, it is protective for the development of ulcerative colitis.

IBD subtypes can be differentiated based on the pattern of inflammation:
• Crohn’s disease is associated with transmural inflammation. Histopathology reveals lymphoid aggregates with noncaseating granulomas.
• Ulcerative colitis is associated with mucosal inflammation (superficial submucosa may also be affected). Histopathology reveals crypt abscesses with neutrophils, but no granulomas.

IBD subtypes can be differentiated based on the affected location within the GI tract:
• Crohn’s disease begins anywhere from the mouth to anus with “skip lesions”.The terminal ileum is most commonly involved while the rectum is rarely involved.
• Ulcerative colitis begins in the rectum (always involved) and can extend up to the cecum, with continuous involvement (and sparing of the remainder of the GI tract).

IBD subtypes can be differentiated based on gross morphological appearance:
• In Crohn disease there is “cobblestone” mucosa, strictures, and creeping fat
• In ulcerative colitis, there are pseudopolyps

IBD subtypes can be differentiated based on imaging with barium contrast:
• Crohn disease may present with “string sign” (due to narrowing of the lumen)
• Ulcerative colitis may present with “lead pipe sign” (loss of haustra)

IBD subtypes can be differentiated based on associations with certain extraintestinal disorders:
• Crohn disease is associated with calcium oxalate kidney stones and gallstones
• Ulcerative colitis is associated with primary sclerosing cholangitis (p-ANCA positive)
Both subtypes are associated (to varying degrees) with the following disorders:
• Pyoderma gangrenosum
• Erythema nodosum
• Ankylosing spondylitis
• Uveitis
• Aphthous ulcers
• Arthritis

Serologic studies for the presence of anti-Saccharomyces cerevisiae antibodies (ASCA) and perinuclear anti-neutrophil cytoplasmic antibodies (p-ANCA) can contribute to the differentiation of IBD subtypes. Helpful patterns are:
• Crohn disease: ASCA-positive, p-ANCA negative
• Ulcerative colitis: ASCA-negative, p-ANCA positive

Systemic complications of both IBD subtypes include:
• Malnutrition due to intestinal inflammation. This is particularly common in patients with Crohn’s disease (which most commonly involves the terminal ileum) since the absorption of both vitamin B12 and bile salts occurs in the terminal ileum.

• Adenocarcinoma of the large colon due to persistent inflammation. This can be seen in both inflammatory bowel diseases but is more common in ulcerative colitis since ulcerative colitis always involves the colon.

The risk of developing inflammatory bowel disease-associated adenocarcinoma is proportional to:
• Disease duration (increased risk at 8-10 years)
• Extent of colonic involvement (increased risk with pancolitis)
• Frequency and intensity of inflammation (increased risk with more intense and frequent relapses)

There are several important GI complications more closely associated with Crohn disease, most notably the following:
• Obstruction caused by stricture formation
• Fistula formation
• Perianal disease
• Cholelithiasis

There are several important GI complications more closely associated with ulcerative colitis, most notably:
• Toxic megacolon
• Sclerosing cholangitis

Cutaneous pathologies that are associated with inflammatory bowel diseases include erythema nodosum and pyoderma gangrenosum.

Pyoderma gangrenosum is a neutrophilic dermatosis that appears as a purulent ulcer with a violaceous edge. The majority of patients with pyoderma gangrenosum have an associated systemic disease, most commonly inflammatory bowel disease.

Treatments for ulcerative colitis include:
• 5-aminosalicylic acid formulations (e.g. sulfasalazine, mesalamine)
• 6-mercaptopurine (purine synthesis inhibitor)
• Infliximab (anti-TNFα antibody)
• Colectomy

Treatments for Crohn disease include:
• Corticosteroids
• Azathioprine (purine synthesis inhibitor)
• Infliximab (anti-TNFα antibody)
• Adalimumab (anti-TNFα antibody)
• Antibiotics (ciprofloxacin, metronidazole)

#inflammatoryboweldisease #clinicalessentials #gastroentrology #pathology #internalmedicine #Crohndisease #Crohnsdisease #ulcerativecolitis

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Addiction needs to be treated like a disease and we need to follow the disease model.

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In this video Chris outlines the basic requirements for employers and employees, starting with the Health and Safety at Work Act 1974, which applies to both employers and employees and also the Management of Health and Safety at Work Regulations 1999.
A few of the thing this will cover:-
1. Risk Assessment
2. PPE
3. COSHH Assessment

If you found this video helpful, we have lots of other videos and free information available on our website, and check out our other videos on the channel.

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A Mount Sinai Morningside and Mount Sinai West Department of Medicine Grand Rounds presented by Charles Branas, PhD, Gelman Professor and Chair, Department of Epidemiology, Columbia University, Mailman School of Public Health

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(USMLE topics, cardiology, blood disorders) Genetics, different forms of SCD, pathophysiology, and treatment. This video is available for instant download licensing here: https://www.alilamedicalmedia.com/-/galleries/all-animations/heart-and-blood-circulation-videos/-/medias/20d89e5d-4da4-4e1d-8f53-5a293088ad11-sickle-cell-disease-narrated-animation
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Sickle cell disease is a group of inherited blood disorders in which the body produces abnormally-shaped red blood cells that look like crescent moons or sickles. Sickle cells have a shorter-than-normal life span; their premature destruction results in shortage of red cells, known as anemia. Signs of anemia include shortness of breath, fatigue, and delayed growth in children. Unlike normal red cells which are pliant, sickle cells are rigid and also sticky. They may clump together and stick to the walls of blood vessels, causing obstruction in small vessels and subsequent reduced oxygen supply to various organs. This happens repeatedly and manifests as periodic episodes of pain, called crises, which can last hours to days, and may result in organ damage, especially in the eyes, lungs, kidneys, bones and brain. The spleen has to handle large numbers of dead red cells and becomes enlarged and fibrous, its immune function declines, making the body more vulnerable to infections. In an attempt to compensate for blood cell loss, the bone marrow tries to produce more cells and grows larger, causing bones to weaken. Other signs include jaundice, a result of rapid destruction of heme.
Hemoglobin is the major component of red blood cells and is responsible for oxygen transport. The adult hemoglobin, or hemoglobin A, is composed of 4 protein chains: 2 alpha and 2 beta. The beta subunit is encoded by the HBB gene. Several mutations in HBB gene are responsible for the disease. Each individual has two copies of HBB gene. The disease develops when both copies are mutated, producing no normal beta globin. The 2 copies may be mutated differently, producing two different forms of abnormal beta subunits in the same person. Various combinations of these mutations produce different forms of sickle cell disease, but the most common and also most severe, called sickle cell anemia, is caused by 2 copies of the same mutation producing the mutated hemoglobin S. Each copy comes from a parent. The 2 parents each carry one copy of the mutated gene, but they typically do not show any symptoms. This pattern of inheritance is called autosomal recessive.
Hemoglobin S has the tendency to form polymers under low oxygen conditions. This process is called sickling, or gelation, for the gel-like consistency of the resulting polymer. As the polymer filaments grow, they eventually involve the cell membrane and distort the cell into the characteristic crescent shape. Apart from oxygen tension, the presence of other hemoglobins also seems to affect the sickling process. Normal adult hemoglobin inhibits sickling and this explains why heterozygous parents, who produce both mutated hemoglobin S and normal hemoglobin A, do not usually develop the disease. Fetal hemoglobin F, which has 2 gamma chains in place of 2 beta chains, also suppresses sickling. Infants born with the condition seem to benefit from high levels of fetal hemoglobin in the first few months of life: they do not develop symptoms until the age of 6 months or so, when fetal hemoglobin levels drop.
Bone marrow transplantation is currently the only known cure for sickle cell disease. It involves replacing the diseased stem cells in the bone marrow with healthy cells from an eligible donor, usually a relative. The procedure however is complex and finding a suitable donor can be difficult. In most cases, treatments aim to avoid crises, relieve symptoms and prevent complications.
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Sickle Cell Anemia. Written by Paulo César Naoum and Alia F. M. Naoum.

A São José do Rio Preto’s Science and Technology Academy video.
www.ciencianews.com.br

Animated by Birdo Studio
www.birdo.com.br

Full Credits:
Production: Birdo
Script: Dr. Paulo César Naoum, Aliá F. M. Naoum
Direction: Luciana Eguti, Paulo Muppet
Storyboard: Paulo Muppet
Design: Rafael Gallardo
Animation and additional design: Antonio Linhares, Fernando Finamore, Pedro Eboli, Rafael Gallardo, William Iamazi Ferro
Sound design: Rafael Gallardo, Antonio Linhares

2011

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